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RanBP9 Overexpression Down-Regulates Phospho-Cofilin, Causes Early Synaptic Deficits and Impaired Learning, and Accelerates Accumulation of Amyloid Plaques in the Mouse Brain.
Authors: Palavicini JP, Wang H, Minond D, Bianchi E, Xu S, Lakshmana MK.
Journal: J Alzheimers Dis.
PubMed ID: 24254706
COPS5 (Jab1) increases β-site processing of amyloid precursor protein and Aβ generation by stabilizing RanBP9 protein levels.
Authors: Hongjie Wang, Debleena Dey, Ivan Carrera, Dmitriy Minond, Elisabetta Bianchi, Shaohua Xu, Madepalli K. Lakshmana.
Journal: J Biol Chem.
PubMed ID: 23926111
Sc65 is a novel endoplasmic reticulum protein that regulates bone mass homeostasis.
Authors: Gruenwald K, Castagnola P, Besio R, Dimori M, Chen Y, Akel NS, Swain FL, Skinner RA, Eyre DR, Gaddy D, Suva LJ, Morello R.
Journal: J Bone Miner Res.
PubMed ID: 23959653
RanBP9 aggravates synaptic damage in the mouse brain and is inversely correlated to spinophilin levels in Alzheimer’s brain synaptosomes.
Authors: Palavicini JP, Wang H, Bianchi E, Xu S, Rao JS, Kang DE, Lakshmana MK.
Journal: Cell Death Dis. 4:e667
PubMed ID: 23764848
Striking reduction of amyloid plaque burden in an Alzheimer's mouse model after chronic administration of carmustine.
Authors: Hayes CD, Dey D, Palavicini JP, Wang H, Patkar KA, Minond D, Nefzi A, Lakshmana MK.
Journal: BMC Med. 11:81
PubMed ID: 23531149
Chronic Cladribine Administration Increases Amyloid Beta Peptide Generation and Plaque Burden in Mice.
Authors: Hayes CD, Dey D, Palavicini JP, Wang H, Araki W, Lakshmana MK.
Journal: PLoS One. 7(10):e45841
PubMed ID: 23056220
BACE1 elevation is involved in amyloid plaque development in the triple transgenic model of Alzheimer's disease: differential Aβ antibody labeling of early-onset axon terminal pathology.
Authors: Cai Y, Zhang XM, Macklin LN, Cai H, Luo XG, Oddo S, Laferla FM, Struble RG, Rose GM, Patrylo PR, Yan XX.
Journal: Neurotox Res. 21(2):160-174
PubMed ID: 21725719

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