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Ginsenoside-Rg1 inhibits endoplasmic reticulum stress-induced apoptosis after unilateral ureteral obstruction in rats.

Ren Fail. 2015-06;

LiSha-sha，YeJian-ming，DengZhi-yong，YuLi-xia，GuXiao-xia，LiuQi-

Products/Services Used	Details	Operation
Polyclonal Antibody Services	… Louis, MO) Mouse anti-CHOP mAb was purchased from cell signaling Technology (Boston, MA) Polyclonal goat anti-glyceraldehyde-3-phosphate dehydrogen- ase (GAPDH) was purchased from GenScript (Piscataway, NJ) …	Get A Quote

摘要

Endoplasmic reticulum (ER) stress and unfolded protein response (UPR) are implicated in many fibrotic diseases， including renal fibrosis. Whether Ginsenoside-Rg1 (G-Rg1) could attenuate renal fibrosis via suppression of ER stress and UPR has not been reported. The aim of this study was to explore the effect of G-Rg1 on ER stress and UPR-induced apoptosis in kidneys with unilateral ureteral obstruction (UUO) rat model.