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Serum copper and ferroportin in monocytes of hemodialysis patients are both decreased but unassociated.

Int Urol Nephrol. 2014; 
Eleftheriadis T, Pissas G, Antoniadi G, Filippidis G, Golfinopoulos S, Spanoulis A, Liakopoulos V, Stefanidis I.
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摘要

Disturbed iron homeostasis contributes to resistance to recombinant human erythropoietin (rHuEpo) in hemodialysis (HD) patients. Although increased hepcidin, which downregulates the iron exporter ferroportin, had been incriminated, such an association has not been confirmed. Albeit not universally accepted, it has been supported that in case of copper deficiency, decreased activity of multicopper oxidases induces endocytosis and degradation of ferroportin. Ferroportin in monocytes, serum copper, ceruloplasmin and markers of iron status were measured, and associations with rHuEpo resistance index (ERI) were evaluated.,After a 4-week washout period from iron treatment, 34 HD patients and 20 healthy volunteers enr... More

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